The researchers therefore administered orally , 3 mg / kg to mice that developed capsaicin many gastrointestinal tumors. Result: taking capsaicin inhibited the activation of the EGF receptor and suppressed the proliferation of cells epithelial. The treatment also allowed the mice to live longer, with a gain of about 30% in the life. The treatment was even more effective when combined with the celecoxib drug anti-inflammatory non-steroidal inhibitor of COX-2, used in the treatment of pain and symptoms of osteoarthritis .
http://androidstars.newsvine.com/_news/2014/08/05/25184616-man-of-flores-was-down-syndrome-the-controversy-continues
http://androidgeek.ucoz.com/blog/the_devil_avoids_sea_waves/2014-08-06-16
http://carmiell.blogspot.com/2014/08/the-epidemic-of-ebola-threat-throughout.html
Therefore, capsaicin eliminate intestinal tumors by stimulating the TRPV1 receptor. For Eyal Raz, "our data suggest that people at high risk of developing recurrent intestinal tumors could benefit from the chronic activation of TRPV1" . The researchers therefore propose to administer TRPV1 agonists (such as capsaicin) with a COX-2 inhibitor to prevent the formation of intestinal carcinoma.
http://androidstars.newsvine.com/_news/2014/08/05/25184616-man-of-flores-was-down-syndrome-the-controversy-continues
http://androidgeek.ucoz.com/blog/the_devil_avoids_sea_waves/2014-08-06-16
http://carmiell.blogspot.com/2014/08/the-epidemic-of-ebola-threat-throughout.html
Therefore, capsaicin eliminate intestinal tumors by stimulating the TRPV1 receptor. For Eyal Raz, "our data suggest that people at high risk of developing recurrent intestinal tumors could benefit from the chronic activation of TRPV1" . The researchers therefore propose to administer TRPV1 agonists (such as capsaicin) with a COX-2 inhibitor to prevent the formation of intestinal carcinoma.
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